U.S. patents available from 1976 to present.
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Pharmaceutical composition for the treatment of diabetes mellitus

Patent 6692777 Issued on February 17, 2004. Estimated Expiration Date: Icon_subject January 22, 2022. Estimated Expiration Date is calculated based on simple USPTO term provisions. It does not account for terminal disclaimers, term adjustments, failure to pay maintenance fees, or other factors which might affect the term of a patent.

Inventor

Application

No. 10/050933 filed on 01/22/2002

US Classes:

424/725PLANT MATERIAL OR PLANT EXTRACT OF UNDETERMINED CONSTITUTION AS ACTIVE INGREDIENT (E.G., HERBAL REMEDY, HERBAL EXTRACT, POWDER, OIL, ETC.)

Examiners

Primary: Witz, Jean C.

Attorney, Agent or Firm

International Classes

A61K 31/60 (20060101)
A61P 3/10 (20060101)
A61P 3/00 (20060101)
A61K 33/24 (20060101)
A61K 33/32 (20060101)
A61K 33/30 (20060101)
A61K 31/4415 (20060101)
A61K 31/506 (20060101)
A61K 31/51 (20060101)

Foreign Application Priority Data

2001-01-22 KR

Abstract

This invention relates to a pharmaceutical composition containing herbal ingredients for the treatment of diabetes mellitus and more particularly, to the antidiabetic composition comprising 1) 15 herbal ingredients (i.e., Shinseng Radix, Coptis Rhizoma, Ligustri Fructus Semen, Salix spp. Cortex, Rhei coreani Rhizoma, Anemarrhena Rhizoma, Salviae Radix, Scrophulariae Radix, Lycii Cortex Radicis, Reynoutriae Radix, Platycodi Radix, Astragali Radix, Puerariae Radix, Atractylis Rhizoma, and Morus alba Radix Cortex), 2) vitamins such as B1 and B6, and 3) zinc, manganese, chromium, germanium as inorganic materials.The antidiabetic herbal composition of this invention for the prevention and treatment of diabetes serves to lower the glucose level in diabetic patients and prevent the destruction of beta-cell in the pancreas, while increasing insulin secretion based on the mechanism of recovering the function of damaged beta-cell. Further, the antidiabetic herbal composition of this invention is quite effective in the treatment of insulin-dependent (type I) diabetes and non-insulin-dependent (type II) diabetes, since it shows the constant therapeutic effect due to better stability of therapeutic effect among individual patients.

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